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A study has shown that the increase of the brain enzyme called puromycin-sensitive aminopeptidase, which is the most abundant brain peptidase seen in mammals, can slow the harmful accumulation of toxic tau proteins, which ultimately
Hyperphosphorylation of the tau protein (tau inclusions, pTau) can result in the self-assembly of tangles of paired helical filaments and straight filaments, which are involved in the pathogenesis of Alzheimer's disease, frontotemporal dementia, and other
Toxic form of tau protein foils memory formation in Alzheimer’s. Study in mice provides a mechanism for how the notorious protein stops neurons from strengthening their connections – and how we form memories